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Furthermore, the Trx system mediates cross‑regulation between autophagy and ferroptosis, as well as autophagy and apoptosis, thereby influencing cellular responses to stress conditions

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This manifests as genomic instability through the accumulation of mutagenic lesions like 8-oxoguanine (8-oxoG), which drives both disease risk and progression by acting as an epigenetic-like mark [230, 231]