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It has been shown to inhibit the TLR4/NF‐κB/NLRP3 pathway, a key receptor in the Toll‐like receptor family that mediates LPS‐stimulated tissue damage, apoptosis, and inflammation, thus protecting VECs and preventing thrombosis and atherosclerosis progression (Liu et al

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An antibody engineered metalloenzyme for mediating cell–cell communication and activation of immuno- and chemotherapy