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Clinical presentation is explained primarily by lack of energy production from long chain fatty acids but with metabolic disturbance confined to the tissues where CPT1A is expressed

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First, while Jnk2 was globally deleted in all cell types, Jnk1 was specifically ablated in hepatocytes but present in non-parenchymal cells, implying an opposing role of Jnk1 in different subsets of liver cells as well as in infiltrated inflammatory cells