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They likely reflect the difference between pathological IGF-1 excess (which does appear to increase cancer risk and accelerate ageing in some models) and normalisation of IGF-1 in individuals whose levels have declined below an optimal physiological range due to somatopause

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BPA also disrupted rodents’ immune function, gastrointestinal tract microbiome, liver, pancreatic β cell function, and the hypothalamic regulation of food intake (Fig