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The molecular mechanism of interaction between the two cells is mainly the binding of p-selectin on activated platelets and PSGL-1 on neutrophils, known as platelet-neutrophil aggregates (PNAs)

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The ability of GH and IGF to modulate key signal transduction pathways such as the MAP kinase/ERK, Jak/STAT, and the PI3K/Akt pathway along with the subsequent effects on cell division and steroidogenesis indicates that these growth factors are centrally located to alter cell fate during proliferation and survival