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Role of receptor for advanced glycation end-products and signalling events in advanced glycation end-product-induced monocyte chemoattractant protein-1 expression in differentiated mouse podocytes

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83 Concurrently, drug-resistant tumor cells upregulate Fabp7 and suppress Lpcat3 via altered H3K27ac/H3K9ac marks, leading to ferroptosis resistance, enhanced FAO, and disrupted circadian signaling in T cells, all of which contribute to immune evasion and therapy failure