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Hepatitis B reactivation and flares have been described following treatment with tumor necrosis factor (TNF)-alpha inhibitors, anti-CD20 agents, corticosteroids, anti-T cell therapy, Janus kinase (JAK) inhibitors, anti-IL6 therapy, and other cytokine and integrin inhibitors.[13,14,15,16,17,18,19,20,21,22,23...] The reactivation risk is highly dependent on the baseline HBsAg status and the specific immunomodulatory medication used.[8] Persons Undergoing Solid Organ or Bone Marrow Transplantation : Hepatitis B reactivation has been well described among individuals who undergo solid organ or bone marrow transplantation, including among HBsAg-negative and anti-HBc-positive patients.[24,25] Hepatitis B reactivation has also been described among individuals who were previously negative for HBV but received a liver transplant from an anti-HBc positive donor.[26,27] Persons Receiving Treatment for HCV : Reactivation of HBV has been increasingly recognized as a potential adverse event associated with HCV direct-acting antiviral (DAA) therapy.[28,29,30,31] In this setting, HBV reactivation has been observed with multiple different DAA regimens.[28,30] Although the risk is highest for HBsAg-positive individuals receiving DAAs, rare cases of reactivation have occurred in persons with isolated anti-HBc.[28,30] The mechanism for HBV reactivation during treatment with HCV DAA treatment remains unknown
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Guillemin R, Brazeau P, Bohlen P, Esch F, Ling N, Wehrenberg WB
