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Its primary mechanisms are: Actin regulation — critical for cell migration and tissue repair Anti-inflammatory signalling through the NF-κB pathway19 Angiogenesis — promoting new vessel formation in damaged tissue20 Cardioprotection — demonstrating direct myocardial repair properties in animal models21 TB-500 Cardiovascular Long COVID: The Strongest Use Case Long COVID cardiovascular sequelae are well-documented: Myocarditis and pericarditis in a subset of patients POTS (postural orthostatic tachycardia syndrome) Reduced VO₂ max and exercise intolerance Microclotting and endothelial damage TB-500's most compelling evidence base is cardiovascular

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