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In summary, GB protects mitochondrial function and reduces mitochondria-dependent neuronal apoptosis by promoting AMPK activation in neuronal cells, specifically upregulating the expression of PINK 1, which provides a novel target for intervention in ischemic stroke therapy ( Jian-Ming Zhou’s team has elucidated a novel anti-inflammatory mechanism of ginkgolides through the oxygen glucose deprivation and reoxygenation (OGD/R) model: significantly inhibiting the release of inflammatory factors from BV2 microglial cells through the targeted modulation of the TLRs/MyD88/NF-κB signaling pathway, and this effect demonstrates specific neuroprotective value in the treatment of ischemic stroke (Zhou et al., 2016) (Figure 1)