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The accumulation of α-syn in DA neurons into higher molecular pathological structures causes neurodegeneration, which further affects autophagy/lysosomal balance, mitochondrial homeostasis, ER, synaptic function, oxidative stress, and excitotoxicity [156]

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(Luo et al., 2022) addressed this by using ECM-modified hydrogels (ECM-Gels) to load cartilage endplate stem cells (CESCs) overexpressing sphingosine kinase 2 (Sphk2)